By Silvano Bonotto, Roland Goutier, René Kirchmann
Biology and Radiobiology of Anucleate structures, I. micro organism and Animal Cells files the lawsuits of the three-day symposium on Biology and Radiobiology of Anucleate structures held in Mol, Belgium on June 21-23, 1971.
This compilation frequently makes a speciality of the anucleate platforms, yet a few papers facing the functionality of membrane-bound polyribosomes and behaviour of remoted mobile organelles also are incorporated.
The subject matters mentioned comprise morphogenesis and synthesis of macromolecules within the absence of the nucleus; construction of DNA-less micro organism; and variations of radiosensitivity in nucleate and anucleate amoeba fragments. The heterogeneity of membrane-bound polyribosomes of mouse myeloma cells in tissue tradition; squid gigantic axon; and cytoplasmic harm resulting in hold up of oral regeneration in Stentor coeruleus also are elaborated. This e-book likewise covers the legislation of protein synthesis in anucleate frog oocytes and DNA, RNA, and protein synthesis in anucleate fragments of sea urchin eggs.
This booklet is an effective reference for college kids and researchers desiring to collect wisdom of the traditional and irradiated mobilephone and sophisticated family among its nucleus and cytoplasm.
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Additional resources for Biology and Radiobiology of Anucleate Systems. Bacteria and Animal Cells
1 - A schematic representation of the morphological alteration of thermosensitive mutants altered in the different steps of cellular division. The open arrow J"L signifies the process(es) altered. The closed arrow ^ - signifies the normal process (es) of cellular division. The genes, and the typical mutations are indicated under the each figures, respectively. 41 Y. HIROTA AND M. RICARD DnaB(T42) PorA(T32) σ α ^ ° ° DnaD(CT7) < - > FtsA(T84) FtsC(MT108) Fig. 2 - A preliminary genetic map of the mutations altered at the different steps of cellular division.
3 of the 12 thermosensitive-mitomycin-resistant mutants were found to segregate DNA-less cells at the non-permissive temperature (Table l a ) . When the original thermosensitive mutant, MFT 100, is grown at 41°C, cell division is arrested, filaments form, and the colony forming capacity is lost exponentially. 4a,4b). They also survive better at 41°C. The chromosome location of the DivC mutation was mapped near the His loci. 33 Y. HIROTA AND M. RICARD Among eight independent mitomycin-resistant mutants of DnaB, all remaining thermosensitive, three were found to segregate DNA-less cells, while the others and the parental strain do not (Table l b ) .
37 Y. HIROTA AND M. S. coli. , 40,885-893. Woody (1961) Resistance and cross-resistance of Escherichia coli mutants to antitumour agent mitomycin C. , 26, 509-520. Cook (1964) Mechanism of action of nadilixic acid on Escherichia coli. , &8, 1112-1118. Hirota,Y. Jacob (1966) Production de bactéries sans DNA. , 262, 1619-1621. coli affected in the processes of DNA synthesis and cellular division. Cold Spring Harbor Symp. , 677-693. Nakai (1968b) On the process of cellular division in Escherichia coli.