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In demand participants deal with matters concerning the toxicity of aluminum which factors an encephalopathy in renal dialysis sufferers and can also be recognized to wreck animals and vegetation through acid rain. Examines the chemistry and biology of aluminum compounds targeting the proof for and opposed to aluminum's position in Alzheimer's affliction.

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The A1 content of bone went up to a certain level and then levelled off; then soft tissue levels of A1 increased. We measured hydroxyproline excretion as an index of bone turnover. We hypothesized that if bone turnover was increased, more Al would be released and hence would move into other tissues. There are several questions. When A1 leaves bone, is it bound to transferrin or to some other protein? Does this affect where it goes? Moreover, would enough A1 be released from bone during turnover to be practically significant?

017 mg/l). 04mg aluminium from water. Food additives as a source of aluminium Food additives are a major source of dietary aluminium in the United States, although aluminium-containing additives are present in only a limited number of foods (Greger 1985, Pennington 1987). Variability in the usage of these foods make it very difficult to estimate aluminium intake from these sources. In 1982 approximately four million pounds weight of aluminium was used in food additives in the United States (Committee on Food Additives Survey Data 1984).

Food Chem Toxicol 21 :103- 109 Lione A 1985 Aluminum intake from non-prescription drugs and sucralfate. Gen Pharmacol 16:223-228 McGraw ME, Bishop N, Jamison R et a1 1986 Aluminum content of milk formulae and intravenous fluids used in infants. Lancet 1:157 Miller RG, Kopfler FC, Kelty KC, Stober JA, Ulmer NS 1984 The occurrence of aluminum in drinking water. J Am Water Assoc 76234-91 Pennington JAT 1987 Aluminum content of foods and diets. Food Addit Contam 5~161-232 Pennington JAT, Jones JW 1989.

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