Download Handbook on Cerebral Venous Thrombosis (Frontiers of by V. Caso, G Agnelli PDF

By V. Caso, G Agnelli

Cerebral veins include approximately 70 in step with cent of the entire cerebral blood quantity, yet cerebral venous thrombosis (CVT) happens a few thousand occasions much less usually than arterial stroke. CVT impacts regularly teenagers and youngsters, and in approximately one area of circumstances the reason is still unknown. Written by means of foreign specialists, this e-book is facing epidemiology, possibility components, coagulation issues, scientific presentation - specially targeting headache, present neuroradiological therapy, issues and long term analysis of CVT. The e-book covers the entire spectrum of CVT pathogenesis and provides a brand new and potent method of enhance previous analysis, realize new chance elements and to spot the main serious manifestations, which require extra competitive treatments.Neurologists, interventional neuroradiologists, neurosurgeons, and all physicians who're excited by the care of sufferers with CVT will welcome this booklet as an invaluable and up to date scientific advisor.

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Additional info for Handbook on Cerebral Venous Thrombosis (Frontiers of Neurology and Neuroscience)

Sample text

Rich C, Gill JC, Wernick S, Konkol RJ: An unusual cause of cerebral venous thrombosis in a fouryear-old child. Stroke 1993;24:603–605. Taha JM, Crone KR, Berger TS, Becket WW, Prenger EC: Sigmoid sinus thrombosis after closed head injury in children. Neurosurgery 1993;32:541–545. Ochagavia AR, Boque MC, Torre C, Alonso S, Sirvent JJ: Dural venous sinus thrombosis due to cranial trauma. Lancet 1996;347:1564. D’Alise MD, Fichtel F, Horowitz M: Sagittal sinus thrombosis following minor head injury treated with continuous urokinase infusion.

Increased blood concentrations of homocysteine have been suggested to be a modifiable, independent risk factor for coronary artery disease, stroke, and DVT. Indeed, two meta-analyses found an association between plasma homocysteine and DVT [146, 147]. With regard to CVST, four case-control studies including, altogether, 222 patients and 472 controls supported the homocysteine hypothesis [148–151]. 001) for CVST in patients with hyperhomocysteinemia [15]. Homocysteine is thought to promote thrombosis through enhanced platelet activation, increased thrombin generation, and impaired fibrinolysis, and by causing endothelial dysfunction [152].

If the pathogenesis and risk factors of venous thromboembolism are less well understood than those of arterial disease, far less information is available for CVST. g. age and obesity) do not play a role in CVST limit our understanding of the disease. Further multicentered, case-controlled studies are crucial for determining the etiology of, and, therefore, methods for the prevention of, CVST. References 1 2 3 4 5 6 7 8 Levine M, Walter S, Lee H, Haines T, Holbrook A, Moyer V: Users’ guides to the medical literature.

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