Liver sickness is a speedily growing to be speciality, and nurses and healthiness care execs have to have the appropriate wisdom and talents to take care of sufferers with liver difficulties in a secure and potent means. Liver Diseases is a complete, evidence-based, sensible advisor to the nursing care and administration of sufferers with liver illness.
Liver Diseases explores a number liver stipulations, together with cirrhosis, portal high blood pressure, alcoholic liver ailment, viral hepatitis, autoimmune hepatitis, Wilson’s illness and acute liver failure. It appears to be like on the anatomy & body structure of the liver, review of liver functionality and diagnostic experiences, acute and protracted liver ailment, being pregnant similar liver sickness, liver transplantation and infectious liver ailments. for every situation the epidemiology, issues and nursing administration are supplied, in addition to illustrative case experiences and extra interpreting. Liver Diseases is a useful instrument for all nurses and future health care pros operating with individuals with liver disease.
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The first united kingdom ebook on care of the liver from a nursing and healthcare perspectiveContent:
Chapter 1 Anatomy and body structure (pages 1–14): Chris Hill
Chapter 2 evaluation of Liver functionality and Diagnostic stories (pages 15–31): Lynda Greenslade
Chapter three Jaundice (pages 32–45): Michelle Clayton
Chapter four Portal high blood pressure (pages 46–60): Dr Terence Wong
Chapter five Ascites, Spontaneous Bacterial Peritonitis, Hyponatraemia and Hepatorenal Failure (pages 61–78): Suzanne Sargent
Chapter 6 Hepatic Encephalopathy (pages 79–92): Catherine Houlston and Helen O'Neal
Chapter 7 Alcohol?Related Liver affliction (pages 93–118): Kerry Webb
Chapter eight Non?Alcoholic Fatty Liver affliction (pages 119–134): Dr Antonis Nikolopoulos and Jude A. Oben
Chapter nine Viral Hepatitis (pages 135–157): Tracey Dudley
Chapter 10 Autoimmune Hepatitis (pages 158–176): Sarah Hughes and Michael Heneghan
Chapter eleven fundamental Biliary Cirrhosis and first Sclerosing Cholangitis (pages 177–192): Danielle Fullwood
Chapter 12 Metabolic Liver sickness (pages 193–213): Rachel Taylor and Teresa Corbani
Chapter thirteen Acute Liver Failure (pages 214–233): Zebina Ratansi
Chapter 14 meals in Liver illness (pages 234–255): Susie Hamlin and Julie Leaper
Chapter 15 Drug?Induced Liver harm (pages 256–270): Suzanne Sargent
Chapter sixteen Pregnancy?Related Liver sickness (pages 271–286): Suzanne Sargent and Michelle Clayton
Chapter 17 Hepatobiliary Malignancies (pages 287–305): Nikie Jervis
Chapter 18 Liver Transplantation (pages 306–328): Wendy Littlejohn and Joanna Routledge
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Additional info for Liver Diseases: An essential guide for nurses and health care professionals
However, elevation can be seen in patients with chronic liver disease which is possibly caused by impaired Kupffer cell function. Martin and Friedman (2004) suggest the pattern of elevation may suggest the aetiology of the underlying liver disease: Elevated IgG – autoimmune hepatitis Elevated IgM – primary biliary cirrhosis IgA – alcoholic liver disease Tests more specific to individual diseases such as viral hepatitis markers, caeruloplasmin (Wilson’s disease), ferritin (hereditary haemochromatosis), alpha-1 antitrypsin (alpha-1-antitrypsin deficiency) and tumour markers, such as alphafetoprotein, are discussed within the relevant chapters.
Blood should be grouped and saved with the facility of compatible blood being available. Informed consent and a patient information sheet are essential for patients undergoing liver biopsy (BSG, 2004). Whether a patient should have an ultrasound scan before the biopsy to rule out any anatomical abnormalities and for patients whom the liver disease cannot be identified, remains an issue for debate (BSG, 2004). g. aspirin and non-steroidal anti-inflammatory drugs (NSAIDs), need to be discontinued a few days prior to the procedure.
This conjugated bilirubin is then excreted into the bile canaliculi. Bacteria in the gut convert bilirubin to stercobilinogen, which then forms stercobilin. Most stercobilin is excreted in the faeces and is responsible for the colour of faeces. Some stercobilin is reabsorbed from the gut and can then be re-excreted by either the liver or kidneys. Liver cirrhosis/fibrosis Liver capacity for regeneration following a single insult is excellent. A hepactectomy may involve the removal of two thirds of the liver, but the remaining hepatocytes can reproliferate to restore the mass of the organ within days to weeks (Guangsheng and Steer, 2006).
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