Download Stem Cells in Animal Species: From Pre-clinic to by Tiziana A.L. Brevini PDF

By Tiziana A.L. Brevini

This quantity makes a speciality of stem mobile learn and illness modeling in non-murine species. The booklet is split into 3 components: Stem Cells for Pre-Clinical versions, Stem Cells in Non-Conventional Species, and Stem phone Banking for the long run. the 1st part offers an summary of the several pre-clinical stem mobilephone types lately created in animal species, together with the porcine version for center failure, iPSC in huge animal species, Duchenne muscular dystrophy and dogs embryo-derived stem cells and modeling for human illnesses. This part additionally discusses the aptitude benefits and functions of those models.

The moment a part of this publication describes contemporary efforts to exploit stem cells for retaining endangered species, together with the snow leopard and coral reefs. From this attitude, stem cells are a useful software to maintain bio-diversities. Frozen cells and gametes may be got from animals liable to extinction or even from microorganisms and corals being affected by heavy adjustments within the eco-system; this can let the cultivation of a new release of stem mobilephone traces and represents a thrilling chance to aid and make sure the conservation of invaluable forms of residing creatures. This dialogue leads simply into the 3rd part, which discusses stem mobilephone banking as a manner of safeguarding those endangered species.

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Science 279:1528–1530 Fougerousse F et al (2007) Phenotypic correction of alpha-sarcoglycan deficiency by intra-arterial injection of a muscle-specific serotype 1 rAAV vector. Mol Ther 15:53–61 Fritz JD, Danko I, Roberds SL, Campbell KP, Latendresse JS, Wolff JA (1995) Expression of deletion-containing dystrophins in mdx muscle: implications for gene therapy and dystrophin function. Pediatr Res 37:693–700 Gabellini D, Green MR, Tupler R (2002) Inappropriate gene activation in FSHD: a repressor complex binds a chromosomal repeat deleted in dystrophic muscle.

Cell 110:339–348 Gabellini D et al (2006) Facioscapulohumeral muscular dystrophy in mice overexpressing FRG1. Nature 439:973–977 Galvez BG et al (2006) Complete repair of dystrophic skeletal muscle by mesoangioblasts with enhanced migration ability. J Cell Biol 174:231–243 Gaschen FP et al (1992) Dystrophin deficiency causes lethal muscle hypertrophy in cats. J Neurol Sci 110:149–159 Gavina M, Belicchi M, Camirand G (2006) VCAM-1 expression on dystrophic muscle vessels has a critical role in the recruitment of human blood-derived CD133+ stem cells after intra-arterial transplantation.

2001); (b) the heart has no intrinsic parenchymal regenerative capacity because it lacks a stem/progenitor cell population able to generate new myocytes. Despite published evidence that this prevalent view was incorrect (Beltrami et al. 2001; Quaini et al. 2002; Urbanek et al. 2003, 2005; Anversa and Nadal-Ginard 2002; Nadal-Ginard et al. 2003), it took the publication of Bergmann et al. in 2009, based on 14C dating in human hearts showing that during a lifetime the human heart renews ~50 % of its myocytes (Bergmann et al.

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